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Solox (Lansoprazole)
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Lansoprazole

Solox helps with different acid-related disorders. It treats heartburn by decreasing the amount of acid produced in the stomach. Solox can also help cure damage which acid can make to your esophagus. Moreover, Solox helps cure duodenal ulcers (ulcers in the first part of the intestine), gastric ulcers (stomach ulcers) and stomach ulcers as a result of continuous use of pain medications called NSAIDs, nonsteroidal anti-inflammatory drugs (aspirin, ibuprofen and naproxen). Solox is available in 3 forms such as Solox Capsules (15 mg, 30 mg), Solox SoluTab Orally Disintegrating Tablets and Solox for Oral Suspension.

Other names for this medication:
Lansoprazole, Agopton, Amarin, Anzo, Aprazol, Bamalite, Compraz, Degastrol, Epicur, Frilans, Gastrazol, Gastrex, Gastride, Gastrolan, Helicol, Ilsatec, Keval, Lancap, Lancid, Lanfast, Lanpro, Lanproton, Lansacid, Lansazol, Lansec, Lanser, Lanso, Lansobene, Lansohexal, Lansol, Lansoloc, Lansomid, Lansopral, Lansoprazol, Lansoprol, Lansoptol, Lansor, Lansox, Lanton, Lanzap, Lanzol, Lanzopral, Lanzoprazol, Lanzor, Lanzostad, Lanzul, Lapol, Lapraz, Laprazol, Larona, Lasgan, Lasoprol, Lazol, Leedom, Lexid, Limpidex, Monolitum, Ogasto, Ogastoro, Ogastro, Opiren, Prazotec, Prevacid, Propump, Prosogan, Protica, Protoner, Refluxon, Refluyet, Solans, Solox, Takepron, Tersen, Trogas, Ulceran, Ulpax, Zolt, Zomel, Zoprol, Zoton, Zotrole,

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In support of a key role for INPP4B in Akt control, INPP4B depletion activated Akt and increased cellular proliferation. One such enzyme, estrogen sulfotransferase, which converts E2 to inactive E2-3 sulfate, has now been shown to be regulated by estrogen in MCF-7 human mammary cancer cells. Hzf encodes a novel protein containing three C(2)H(2)-type zinc fingers predominantly expressed in megakaryocytes and CFU-GEMM. Growth of these MUC1-transfected cells in type I collagen gels produced marked gel contraction that could be significantly reduced by the synthetic peptide SRGDTG or by growth in serum and fibronectin-depleted media. Based on the success of our recently developed strategy for furan-based crosslinking in DNA duplexes, we here investigated for the first time the use of the furan-oxidation crosslink methodology for the covalent locking of triplex structures by an interstrand crosslink.

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A 43-year-old female showing signs and symptoms of subacute thyroiditis was referred to our hospital for further evaluation of her thyroid function because TSH-binding inhibitor immunoglobulins (TBII) were detected in her serum. Changes in the suicide rate of teenagers and young adults internationally from 1970 to 1980 were examined. This gene, whose localization has been assigned to the short arm of chromosome 11 (position 11p15), is transcribed in HeLa cells as one major messenger RNA, which encodes a 67-residue polypeptide (7645 Da) that shares strong homologies with the corresponding subunits of other eukaryotic and archaeal RNA polymerase subunits.

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Polarized secretion of tissue-plasminogen activator in cultured thyroid cells. The influence of zinc on the blood serum of cadmium-treated rats through the rheological properties. Effects of Salt Concentrations and Nitrogen and Phosphorus Starvations on Neutral Lipid Contents in the Green Microalga Dunaliella tertiolecta.

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Time course of changes in the concentrations of monoamines in the brain structures of pentylenetetrazole-kindled rats. Cytotoxicity and mutagenicity of 6-aminonicotinamide in Chinese hamster ovary cells. Effects of soil water content on the external exposure of fauna to radioactive isotopes. Specifically, the performance of the probe is evaluated by comparison of signal-to-noise (SNR) performance with that of a copper Helmholtz pair, analysis of B1 field homogeneity, and quantification of thermal stability. In contrast, the number of neurons and the total process length of neurons were not affected by 5 alpha-DHT.

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Diazinon, an organophosphate pesticide, induces oxidative stress and genotoxicity in cells deriving from large intestine. Against this backdrop of subclinical iron deficiency, curcumin exerted profound 2 effects on systemic iron, inducing a dose-dependent decline in hematocrit, hemoglobin, serum iron, and transferrin saturation, the appearance of microcytic anisocytotic red blood cells, and decreases in spleen and liver iron content.

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